Glomerulopathies: Nephrotic and Nephritic Syndromes | High Specialty Nephrology

Internal Medicine · Clinical Specialties

How it begins

Una aproximación de nivel experto a las entidades más complejas de la nefrología. Desde las bases moleculares de la barrera de filtración hasta las guías KDIGO más recientes para el tratamiento de las glomerulopatías nefróticas y nefríticas primarias y secundarias, pasando por la interpretación ultraestructural y el análisis del sedimento de orina de alta resolución.

What it covers

1. Ultrastructural Anatomy of the Glomerular Filtration Barrier
2. Load and Size Selectivity in Glomerular Filtration
3. · 1. Size Selectivity
4. · 2. Charge Selectivity
5. Pathophysiology of Nephrotic Syndrome: Massive Proteinuria
6. · The Physiopathological Mechanism of Proteinuria
7. Metabolic and Clinical Consequences of Nephrotic Syndrome
8. · 1. Pathophysiology of Nephrotic Edema
9. · 2. Dyslipidemia, Hyperlipidemia and Lipiduria
10. · 3. Hypercoagulable State and Thromboembolic Risk
11. · 4. Extreme Susceptibility to Infections
12. Etiological Classification of Nephrotic Glomerulopathies
13. Minimal Change Disease (MCD)
14. · Etiology and Molecular Pathogenesis
15. · Histopathology Characteristic
16. · Treatment and Clinical Course
17. Focal and Segmental Glomerulosclerosis (FSGS)
18. · Etiology and Pathophysiological Classification
19. · Histopathology: Columbia Classification
20. · Treatment of Primary FSGS
21. Membranous Nephropathy (NM)
22. · Molecular Immunopathogenesis and Autoantibodies
23. · Ehrenreich and Churg stages by Electron Microscopy
24. · Treatment Based on KDIGO Risk Stratification
25. Membranoproliferative glomerulonephritis (MPGN)
26. · Complement-Based Pathophysiological Reclassification
27. · Immunopathology of C3 Glomerulopathy and C3 Nephritic Factor
28. · Histological Appearance by MO: The Membranoproliferative Pattern
29. Extreme Secondary Causes: Renal Amyloidosis and Diabetic Nephropathy
30. · 1. Renal Amyloidosis (AL/AA)
31. · 2. Diabetic Nephropathy (DN): The Metabolic and Hemodynamic Damage Model
32. Pathophysiology of Nephritic Syndrome: Inflammatory Glomerular Damage
33. · The Mechanism of Glomerular Inflammatory Damage
34. Poststreptococcal Glomerulonephritis (PEGN)
35. · Etiology and Immunopathogenesis
36. · Histological Findings and Immunofluorescence
37. · Clinical Evolution and Complement Profile
38. IgA nephropathy (Berger's disease)
39. · Immunopathogenesis: Four-Hit Hypothesis
40. · Oxford Histological Classification (MEST-C Score)
41. · Treatment of IgA Nephropathy
42. Rapidly Progressing Glomerulonephritis (RPGN)
43. · Pathophysiology of Crescent Formation
44. · Immunopathological Classification of PRGN
45. Pauciimmune Vasculitis Associated with ANCA
46. · Immunological Mechanisms of Neutrophil Activation
47. · ANCA Vasculitis Induction Treatment (PEXIVAS and ADVOCATE Trials)
48. Anti-Glomerular Basal Membrane Antibody Disease
49. · Goodpasture's Autoantigen
50. · Emergency Treatment
51. Lupus Nephritis: Immunopathology and Classification
52. · Immunological Pathophysiology of Lupus Nephritis
53. · ISN/RPS Histological Classification (2018 Revision)
54. · Updated Treatment of Proliferative Classes (III/IV)
55. Glomerular Thrombotic Microangiopathies (TMA)
56. · Pathophysiology of Glomerular Endothelial Damage
57. · Clinical and Etiological Subtypes
58. · Specific Treatment: C5 blockade with Eculizumab
59. Precision Analysis of Urinary Sediment
60. · Sediment Differentiation: Nephrotic Pattern vs. Nephritic Pattern
61. Precision Renal Biopsy and Decision Algorithms KDIGO
62. · Integrated Anatomopathological Processing and Analysis
63. · Therapeutic Decision Algorithm in Glomerulopathies (KDIGO 2021)