Intensive Medicine — Renal, Hydroelectrolyte and Acid-Base Disorders

Intensive Medicine · Clinical Specialties

How it begins

Un estudio exhaustivo y riguroso de los mecanismos homeostáticos, moleculares y biofísicos que rigen la función renal y el equilibrio interno en pacientes en estado crítico, integrando algoritmos terapéuticos avanzados y monitorización de alta definición.

What it covers

1. Kidney and Acid-Base Disorders
2. Kidney Pathophysiology in the Critically Ill Patient
3. · Glomerular Autoregulation Mechanisms
4. · Medullary Hypoxia: An Anatomical Physiological Trap
5. Acute Kidney Failure (AKI): Classification and Biomarkers
6. · Evolution of AKI Clinical Classifications
7. · Critical Limitations of Serum Creatinine in Critical Care Patients
8. · New Generation Biomarkers: Damage vs. Function
9. Acute Tubular Necrosis (ATN) vs. Prerenal AKI
10. · Key Pathophysiological Differentiation
11. · Urinary Indices and their Physiological Basis
12. · Microscopic Analysis of Urinary Sediment
13. Continuous Renal Replacement Therapy (CRRT) in the ICU
14. · Biophysical Solute Transport Mechanisms
15. · Adsorption: The Third Mechanism
16. · CRRT Standard Modalities
17. · Prescription and Metabolic Dosage
18. · Regional Anticoagulation with Citrate (RCA)
19. Sodium Disorders I: Hyponatremia in the ICU
20. · Diagnostic Algorithm Based on Tonicity
21. · Brain Cellular Adaptation to Hyponatremia
22. · Management of Acute Severe Symptomatic Hyponatremia
23. Sodium Disorders II: Hypernatremia in the ICU
24. · Physiopathological Classification according to Volemia
25. · Security Replacement and Correction Strategies
26. Potassium Physiology and Disorders
27. · Hyperkalemia: Immediate Cardiac Threat
28. · Emergency Treatment of Severe Hyperkalemia
29. · Hypokalemia: Pathophysiology and Disorder of the ROMK Channel
30. Calcium, Phosphorus and Magnesium Disorders in Critical
31. · Calcium Disorders: Ionized Fraction and Albumin Correction
32. · Phosphorus Disorders: The Feedback Syndrome and Diagrammatic Failure
33. Advanced Acid-Base Physiology: Henderson-Hasselbalch Approach vs. Stewart
34. · The Classical Henderson-Hasselbalch Hypothesis and its Limitations
35. · Peter Stewart's Physicochemical Approach
36. Metabolic acidosis in the critically ill patient
37. · Categorization by Anion Gap
38. · Elevated Anion Gap Acidosis (Normochloremic Acidosis)
39. · Other Causes of High Anion Gap
40. · Normal Anion Gap Acidosis (Hyperchloremic Acidosis)
41. Metabolic Alkalosis and Acid-Base Respiratory Disorders
42. · Metabolic Alkalosis: Classification Based on Urinary Chlorine
43. · Acid-Base Disorders of Respiratory Origin
44. Mixed Acid-Base Disorders in Intensive Care
45. · Systematic Diagnostic Interpretation Algorithm
46. Cardiorenal Syndrome and Hepatorrenal Syndrome in the ICU
47. · Cardiorenal Syndrome (CRS): Bidirectional Cross Damage
48. · Hepatorrenal Syndrome (HRS / HRS-AKI)
49. · Updated Diagnostic Criteria of the ICA-HRS (International Club of Ascites)
50. Renal Pharmacology and Drug Adjustment in the Critical Patient
51. · Modified Pharmacokinetics in AKI and CRRT
52. · Directed Nephrotoxicity: Common Medications in the ICU
53. · Contrast-Induced Acute Kidney Injury (AKI-CI)