Neurocognitive Disorders — Expert Clinical Atlas

Psychiatry · Clinical Specialties

How it begins

Los Trastornos Neurocognitivos (TNC) constituyen el único grupo de trastornos del DSM-5-TR cuya psicopatología subyacente es, en la mayoría de los casos, conocida y demostrable: representan un declive adquirido respecto a un nivel previo de funcionamiento en uno o más dominios cognitivos, secundario a una alteración estructural, metabólica, vascular, infecciosa, traumática o degenerativa del sistema nervioso…

What it covers

1. Introduction and Concept of Neurocognitive Disorder
2. · 1.1 The conceptual transition: why “dementia” was abandoned as an axis
3. · 1.2 Diagnostic structure in two axes
4. · 1.3 General epidemiology
5. · 1.4 Clinical importance of the category
6. The Six Neurocognitive Domains
7. · 2.1 Complex care
8. · 2.2 Executive function
9. · 2.3 Learning and memory
10. · 2.4 Language
11. · 2.5 Perceptual-motor (visuospatial and praxias)
12. · 2.6 Social cognition
13. DSM-5-TR and ICD-11 Classification
14. · 3.1 The three nuclear syndromes
15. · 3.2 Recognized etiologies
16. · 3.3 Severity and behavior specifiers
17. · 3.4 Comparison with ICD-11
18. Delirium (Acute Confusional Syndrome)
19. · 4.1 Subtypes according to psychomotor activity
20. · 4.2 Predisposing and precipitating factors
21. · 4.3 Delirium vs. Dementia: the crucial differential diagnosis
22. · 4.4 Screening tools
23. · 4.5 Management of delirium
24. Slight and Major TNC: the Specter of Decline
25. · 5.1 Mild Neurocognitive Disorder (mild NCD / MCI)
26. · 5.2 Major Neurocognitive Disorder (dementia)
27. · 5.3 Differential diagnosis with depressive pseudodementia
28. Alzheimer's disease
29. · 6.1 Neuropathology and pathophysiology
30. · 6.2 Risk factors
31. · 6.3 Clinical presentation and evolution
32. · 6.4 Diagnostic criteria and biomarkers
33. · 6.5 Treatment
34. Vascular Neurocognitive Disorder
35. · 7.1 Distinctive clinical characteristics
36. · 7.2 Diagnostic criteria
37. · 7.3 Subtypes and hereditary forms
38. · 7.4 Risk factors and prevention
39. TNC by Lewy Bodies
40. · 8.1 The four nuclear clinical characteristics
41. · 8.2 The “one year” rule and diagnosis
42. · 8.3 Supportive and dysautonomia traits
43. · 8.4 Treatment
44. Frontotemporal Lobar Degeneration
45. · 9.1 Behavioral variant (bcFTD)
46. · 9.2 Primary progressive aphasias (language variants)
47. · 9.3 Neuropathology and overlaps
48. · 9.4 Treatment
49. Parkinson's, Huntington's and Subcortical Dementia
50. · 10.1 Parkinson's disease dementia
51. · 10.2 Huntington's disease
52. · 10.3 Progressive supranuclear palsy and corticobasal degeneration
53. TNC Secondary and Treatable Causes
54. · 11.1 The battery of reversible causes
55. · 11.2 Normal pressure hydrocephalus
56. · 11.3 Alcoholic dementia and Wernicke-Korsakoff syndrome
57. Trauma, Prions and HIV Infection
58. · 12.1 TNC due to brain trauma
59. · 12.2 TNC by prions (Creutzfeldt-Jakob)
60. · 12.3 HIV-associated neurocognitive disorder (HAND)
61. Evaluation and Diagnosis
62. · 13.1 The anamnesis and the informant
63. · 13.2 Cognitive screening tests
64. · 13.3 Laboratory study
65. · 13.4 Neuroimaging
66. · 13.5 Integrated diagnostic algorithm
67. Behavioral and Psychological Symptoms (BPSD)
68. · 14.1 SCPD Spectrum
69. · 14.2 SCPD evaluation
70. · 14.3 Management: non-pharmacological first
71. · 14.4 Pharmacological options (when necessary)
72. Treatment, Care and Prevention
73. · 15.1 General principles of treatment
74. · 15.2 Non-pharmacological interventions
75. · 15.3 The caregiver and planning
76. · 15.4 Prevention: modifiable risk factors
77. Integrative Clinical Synthesis
78. · 16.1 The questions that order the diagnosis
79. · 16.2 The clinical pattern as an etiological compass
80. · 16.3 Clinical errors to avoid
81. · 16.4 Final synthesis