Gastric bazuqueo
Specialty: Gastrointestinal.
Why it occurs
- Obstruction of the gastric outlet tract (pyloric stenosis secondary to scars from recurrent duodenal ulcers or adenocarcinoma of the gastric antrum)
- Severe gastroparesis of diabetic, idiopathic or post-surgical origin due to injury to the branches of the vagus nerve
- Intrinsic or extrinsic duodenal stenosis or compression (aortomesenteric clamp syndrome or Wilkie syndrome)
- Proximal jejunal mechanical obstruction due to flanges or intestinal intussusception.
Initial workup
Physical abdominal succussion maneuver (epigastric auscultation while the patient's pelvis is rocked laterally; positive if a liquid splash is heard more than 4-6 hours after the last fast) | Upper gastrointestinal endoscopy (EDA, after decompression with a large caliber nasogastric tube to avoid bronchoaspiration) | Abdominal CT with oral and intravenous contrast to define the anatomy of the pyloric area | Esophagogastroduodenal transit with water-soluble contrast to detect delayed emptying.
red flags
Recurrent vomiting of poorly digested food eaten more than 12 hours before, severe dehydration, hypochloremic metabolic alkalosis with severe hypokalemia, rapid and involuntary weight loss and progressive cachexia.
Standard management
- Intravenous prokinetics — metoclopramide 10 mg IV every 8 hours or erythromycin 200 mg IV slowly infused to force emptying in gastroparesis, formally contraindicated if there is complete mechanical pyloric obstruction
- Intravenous hydroelectrolyte replacement fluid therapy — 0.9% saline supplemented with potassium chloride [KCl] according to ionogram to correct metabolic alkalosis and hypokalemia
- Proton pump inhibitors (pantoprazole 40 mg IV every 12 hours to reduce basal gastric secretory volume).
Educational guidance for study. It is NOT a prescription recommendation. The actual choice depends on the cause, the patient, and current guidelines.
- Area
- Gastrointestinal
- Listed causes
- 4
- Treatment options
- 3