Hyperactive borborygmi
Specialty: Gastrointestinal.
Why it occurs
- Fight phase in incipient mechanical intestinal obstruction (peristalsis increases to overcome a physical obstacle)
- Acute infectious or alimentary gastroenteritis with reflex hypermotility due to bacterial or viral enterotoxins
- Malabsorption or feeding intolerance with hypermotility induced by intraluminal osmotic gradient
- Irritable bowel syndrome with a diarrheal pattern due to colonic hyperreactive autonomic stimulation.
Initial workup
Systematic abdominal auscultation to characterize the frequency and tone of noises | Plain x-ray of the abdomen in standing position (to assess air-fluid levels and struggle handles) | Abdominal CT with intravenous contrast if there is suspicion of high or low mechanical obstruction | Emergency analysis: blood count, electrolytes, lactate blood gases and CRP.
red flags
High-frequency metallic intestinal noises followed by absolute abdominal silence (a sign of paralytic ileus due to peritonitis or perforation), intense colic pain coinciding with borborygmus, bilious or fecaloid vomiting, inability to channel gases and stools.
Standard management
- Antispasmodics in gastroenteritis or IBS without suspicion of mechanical obstruction — butylscopolamine bromide 10-20 mg orally or intravenously, or otilonium bromide 40 mg orally to slow coordinated smooth muscle contractions
- Loperamide — 2 mg after each liquid stool in acute non-infectious diarrhea to delay transit, formally contraindicated if there is suspicion of infection by invasive pathogens with fever or dysentery
- Oral rehydration solutions to compensate for water losses caused by hypermotility.
Educational guidance for study. It is NOT a prescription recommendation. The actual choice depends on the cause, the patient, and current guidelines.
- Area
- Gastrointestinal
- Listed causes
- 4
- Treatment options
- 3